[1] 韩萨茹拉.成纤维细胞生长因子(FGF)研究进展.安徽农业科学, 2009, 37(7):3008-3010. SA Ru-la. Review on fibroblast growth factor (FGF). Journal of Anhui Agri Sci, 2009,37(7):3008-3010.
[2] Nunes Q M, Li Y, Sun C, et al. Fibroblast growth factors as tissue repair and regeneration therapeutics. Peerj, 2015, 4(5):e1535.
[3] Kan S H, Elanko N, Johnson D, et al. Genomic screening of fibroblast growth-factor receptor 2 reveals a wide spectrum of mutations in patients with syndromic craniosynostosis. Am J Hum Genet, 2002,70(2):472-486.
[4] Teven C M, Farina E M, Rivas J, et al. Fibroblast growth factor (FGF) signaling in development and skeletal diseases. Genes Dis, 2014, 1(2):199-213.
[5] Yan D, Chen D, Cool S M, et al. Fibroblast growth factor receptor 1 is principally responsible for fibroblast growth factor 2-induced catabolic activities in human articular chondrocytes. Arthritis Res Ther, 2011, 13(4):130.
[6] Harada S, Rodan G A. Control of osteoblast function and regulation of bone mass. Nature, 2003, 423(6937):349-355.
[7] Zou W, Izawa T, Zhu T, et al. Talin1 and Rap1 are critical for osteoclast function. Mol Cell Biol, 2013, 33(4):830-844.
[8] Lu X, Su N, Yang J, et al. Fibroblast growth factor receptor 1 regulates the differentiation and activation of osteoclasts through Erk1/2 pathway. Biochem Biophys Res Commun, 2009, 390(3):494-499.
[9] Soung D Y, Kalinowski J, Baniwal S K, et al. Runx1-mediated regulation of osteoclast differentiation and function. Mol Endocrinol, 2014, 28(4):546-553.
[10] Mirza M A, Karlsson M K, Mellstr m D, et al. Serum fibroblast growth factor-23(FGF-23) and fracture risk in elderly men. Bone Miner Res, 2011, 26(4):857-864.
[11] Wang H, Yoshiko Y, Yamamoto R, et al. Overexpression of fibroblast growth factor 23 suppresses osteoblast differentiation and matrix mineralization in vitro. Bone Miner Res, 2008, 23(6):939-948.
[12] Shalhoub V, Ward S C, Sun B, et al. Fibroblast growth factor 23(FGF23) and alpha-klotho stimulate osteoblastic MC3T3.E1 cell proliferation and inhibit mineralization. Calcif Tissue Int, 2011, 89(2):140-150.
[13] Rhee Y, Bivi N, Farrow E, et al. Parathyroid hormone receptor signaling in osteocytes increases the expression of fibroblast growth factor-23 in vitro and in vivo. Bone, 2011, 49(4):636-643.
[14] Raimann A, Ertl D A, Helmreich M, et al. Fibroblast growth factor 23 and Klotho are present in the growth plate. Connect Tissue Res, 2013, 54(2):108-117.
[15] Wei W, Dutchak P A, Wang X, et al. Fibroblast growth factor 21 promotes bone loss by potentiating the effects of peroxisome proliferator-activated receptor γ. Proc Natl Acad Sci U S A, 2012, 109(8):3143-3148.
[16] Zamli Z, Robson B K, Tarlton J F, et al. Subchondral bone plate thickening precedes chondrocyte apoptosis and cartilage degradation in spontaneous animal models of osteoarthritis. Biomed Res Int, 2014, 2014(16):751-759.
[17] Im H J, Li X, Muddasani P, et al. Basic fibroblast growth factor accelerates matrix degradation via a neuro-endocrine pathway in human adult articular chondrocytes. Cell Physiol, 2008, 215(2):452-463.
[18] Ellman M B, Yan D, Ahmadinia K, et al. Fibroblast growth factor control of cartilage homeostasis. Cell Biochem, 2013, 114(4):735-742.
[19] Im H J, Muddasani P, Natarajan V, et al. Basic fibroblast growth factor stimulates matrix metalloproteinase-13 via the molecular cross-talk between the mitogen-activated protein kinases and protein kinase C delta pathways in human adult articular chondrocytes. Biol Chem, 2007, 282(15):11110-11121.
[20] Schmal H, Zwingmann J, Fehrenbach M, et al. bFGF influences human articular chondrocyte differentiation. Cytotherapy, 2007, 9(2):184-193.
[21] Sonal D. Prevention of IGF-1 and TGFbeta stimulated type Ⅱ collagen and decorin expression by bFGF and identification of IGF-1 mRNA transcripts in articular chondrocytes. Matrix Biol, 2001, 20(4):233-242.
[22] Zhang X, Ibrahimi O A, Olsen S K, et al. Receptor specificity of the fibroblast growth factor family. The complete mammalian FGF family. Biol Chem, 2006, 281(23):15694-15700.
[23] Chia S L, Sawaji Y, Burleigh A, et al. Fibroblast growth factor 2 is an intrinsic chondroprotective agent that suppresses ADAMTS-5 and delays cartilage degradation in murine osteoarthritis. Arthritis Rheum, 2009, 60(7):2019-2027.
[24] Kaul G, Cucchiarini M, Arntzen D, et al. Local stimulation of articular cartilage repair by transplantation of encapsulated chondrocytes overexpressing human fibroblast growth factor 2(FGF-2) in vivo. Gene Med, 2006, 8(1):100-111.
[25] Hunter D J. Pharmacologic therapy for osteoarthritis——the era of disease modification. Nat Rev Rheumatol, 2011, 7(1):13-22.
[26] Ellsworth J L, Berry J, Bukowski T, et al. Fibroblast growth factor-18 is a trophic factor for mature chondrocytes and their progenitors. Osteoarthritis Cartilage, 2002, 10(4):308-320.
[27] Lohmander L S, Hellot S, Dreher D, et al. Intraarticular sprifermin (recombinant human fibroblast growth factor 18) in knee osteoarthritis:a randomized, double-blind, placebo-controlled trial. Arthritis Rheumatol, 2014, 66(7):1820-1831.
[28] Ohbayashi N, Shibayama M, Kurotaki Y, et al. FGF18 is required for normal cell proliferation and differentiation during osteogenesis and chondrogenesis. Genes Dev, 2002, 16(7):870-879.
[29] Liu Z, Xu J, Colvin J S, et al. Coordination of chondrogenesis and osteogenesis by fibroblast growth factor 18. Genes Dev, 2002, 16(7):859-869.
[30] Shimoaka T, Ogasawara T, Yonamine A, et al. Regulation of osteoblast, chondrocyte, and osteoclast functions by fibroblast growth factor (FGF)-18 in comparison with FGF-2 and FGF-10. Biol Chem, 2002, 277(9):7493-7500.
[31] Ellsworth J L, Berry J, Bukowski T, et al. Fibroblast growth factor-18 is a trophic factor for mature chondrocytes and their progenitors. Osteoarthritis Cartilage, 2002, 10(4):308-320.
[32] Whitsett J A, Clark J C, Picard L, et al. Fibroblast growth factor 18 influences proximal programming during lung morphogenesis. Biol Chem, 2002, 277(25):22743-22749.
[33] Moore E E, Bendele A M, Thompson D L, et al. Fibroblast growth factor-18 stimulates chondrogenesis and cartilage repair in a rat model of injury-induced osteoarthritis. Osteoarthritis Cartilage, 2005, 13(7):623-631.
[34] Mithoefer K, Williams R J, Warren R F, et al. The microfracture technique for the treatment of articular cartilage lesions in the knee. A prospective cohort study. Bone Joint Surg Am, 2005, 87(9):1911-1920.
[35] Power J, Hernandez P, Guehring H, et al. Intra-articular injection of rhFGF-18 improves the healing in microfracture treated chondral defects in an ovine model. Orthop Res, 2014, 32(5):669-676.
[36] Barr L, Getgood A, Guehring H, et al. The effect of recombinant human fibroblast growth factor-18 on articular cartilage following single impact load. Orthop Res, 2014, 32(7):923-927.
[37] Reinhold M I, Abe M, Kapadia R M, et al. FGF18 represses noggin expression and is induced by calcineurin. Biol Chem, 2004, 279(37):38209-38219.
[38] Valverde-Franco G, Binette J S, Li W, et al. Defects in articular cartilage metabolism and early arthritis in fibroblast growth factor receptor 3 deficient mice. Hum Mol Genet, 2006, 15(11):1783-1792.
[39] Eswarakumar V P, Monsonego-Ornan E, Pines M, et al. The Ⅲc alternative of Fgfr2 is a positive regulator of bone formation. Development, 2002, 129(16):3783-3793.
[40] Ornitz D M, Marie P J. FGF signaling pathways in endochondral and intramembranous bone development and human genetic disease. Genes Dev, 2002, 16(12):1446-1465.
[41] Ellman M B, Yan D, Ahmadinia K, et al. Fibroblast growth factor control of cartilage homeostasis. Cell Biochem, 2013, 114(4):735-742.
[42] Ohbayashi N, Shibayama M, Kurotaki Y, et al. FGF18 is required for normal cell proliferation and differentiation during osteogenesis and chondrogenesis. Genes Dev, 2002, 16(7):870-879.
[43] Liu Z, Xu J, Colvin J S, et al. Coordination of chondrogenesis and osteogenesis by fibroblast growth factor 18. Genes Dev, 2002, 16(7):859-869.
[44] Bosetti M, Leigheb M, Brooks R A, et al. Regulation of osteoblast and osteoclast functions by FGF-6. Cell Physiol, 2010, 225(2):466-471.
[45] Uchii M, Tamura T, Suda T, et al. Role of fibroblast growth factor 8(FGF8) in animal models of osteoarthritis. Arthritis Res Ther, 2008, 10(4):1-10.
[46] Hung I H, Yu K, Lavine K J, et al. FGF9 regulates early hypertrophic chondrocyte differentiation and skeletal vascularization in the developing stylopod. Dev Biol, 2007, 307(2):300-313. |