| [1] Beenken A, Mohammadi M. The FGF family:biology, pathophysiology and therapy. Nature Reviews Drug Discovery, 2009, 8(3):235-253.
[2] Lee C H, Umemori H. Suppression of epileptogenesis-associated changes in response to seizures in FGF22-deficient mice. Frontiers in Cellular Neuroscience, 2013, 7(2):43.
[3] Jarosz M, Robbez-Masson L, Chioni A M, et al. Fibroblast growth factor 22 is not essential for skin development and repair but plays a role in tumorigenesis. PLoS One, 2012, 7(6):e39436.
[4] Miyake A, Itoh N. Fgf22 regulated by Fgf3/Fgf8 signaling is required for zebrafish midbrain development. Biology Open, 2013, 2(5):515-524.
[5] Umemori H, Fox M A. Fibroblast growth factor 22 contributes to the development of retinal nerve terminals in the dorsal lateral geniculate nucleus. Frontiers in Molecular Neuroscience, 2012, 4:61.
[6] Williams A J, Yee P, Smith M C, et al. Deletion of fibroblast growth factor 22(FGF22) causes a depression-like phenotype in adult mice. Behavioural Brain Research, 2016, 307(Suppl C):11-17.
[7] Itoh N, Ornitz D M. Evolution of the Fgf and Fgfr gene families. Trends Genet, 2004,20(11):563-569.
[8] Olsen S K, Garbi M, Zampieri N, et al. Fibroblast growth factor (FGF) homologous factors share structural but not functional homology with FGFs. The Journal of Biological Chemistry, 2003, 23(36):34226-34236.
[9] Mohammadi M, Olsen S K, Ibrahimi O A. Structural basis for fibroblast growth factor receptor activation. Cytokine Growth Factor Reviews, 2005, 16(2):107-137.
[10] Johnson D E, Lu J, Chen H, et al. The human fibroblast growth factor receptor genes:a common structural arrangement underlies the mechanisms for generating receptor forms that differ in their third immunoglobulin domain. Molecular and Cellular Biology, 1991, 11(9):4627-4634.
[11] Wang F, Kan M, Yan G, et al. Alternately spliced NH2-terminal immunoglobulin-like loop I in the ectodomain of the fibroblast growth factor (FGF) receptor 1 lowers affinity for both heparin and FGF-1. Journal of Biological Chemistry, 1995, 270(17):10231-10235.
[12] Zhang X, Ibrahimi O A, Olsen S K, et al.Receptor specificity of the fibroblast growth factor family, the complete mammalian FGF family. Journal of Biological Chemistry, 2006, 281(23):15694-15700.
[13] Semah F, Picot M C, Adam C, et al. Is the underlaying cause of epilepsy a majorprognostic factor for recurrence? Neurology, 1998, 51(5):1256-1262.
[14] Brodie M J. Diagnosing and predicting refractory epilepsy. Acta Neurologica Scandinavica, 2005, 112(181):36-39.
[15] French J A. Refractory epilepsy:clinical overview. Epilepsia. Series 4, 2007, 48(1):3-7.
[16] Akiko T, Venkatesh J, Erin M, et al. Distinct FGFs promote differentiation of excitatory and inhibitory synapses. Natrue, 2010, 465(7299):783-787.
[17] Nakatake Y, Hoshikawa M, Asaki T, et al. Identification of a novel fibroblast growth factor, FGF-22, preferentially expressed in the inner root sheath of the hair follicle. Biochim Biophys Acta, 2001, 1517(3):460-463.
[18] 赵辩. 临床皮肤病学. 第3版.南京:江苏科技出版社, 2001:1111-1125. Zhao B. Clinical Dermatology. 3rd ed. Najing:Jiangsu Science and Technology Press, 2001:1111-1125.
[19] Beyer T A, Werner S, Dickson C, et al. Fibroblast growth factor 22 and its potential role during skin development and repair. Experimental Cell Research, 2003, 287(2):228-236.
[20] Braun S, Hanselmann C, Gassmann M G, et al. Nrf2 transcription factor, a novel target of keratinocyte growth factor action which regulates gene expression and inflammation in the healing skin wound. Molecular and Cellular Biology, 2002, 22(15):5492-5505.
[21] Braun S, Keller U, Steiling H, et al. Fibroblast growth factors in epithelial repair and cytoprotection. Philosophical Transactions of the Royal Society. Series B:Biological Sciences, 2004, 359(1445):753-757.
[22] Kumin A, Huber C, Rulicke T, et al. Peroxiredoxin 6 is a potent cytoprotective enzyme in the epidermis. American Journal of Pathology, 2006, 169(4):1194-1205.
[23] Keller U, Huber M, Beyer T A, et al. Nrf transcription factors in keratinocytes are essential for skin tumor prevention but not for wound healing. Mol Cell Biol, 2006, 26(10):3773-3784.
[24] Ferrari A J, Charlson F J, Norman R E, et al. The epidemiological modelling of major depressive disorder:application for the global burden of disease study 2010. PLoS One, 2013, 8(7):e69637.
[25] Elhai J D, Contractor A A, Tamburrino M, et al. Structural relations between DSM-5 PTSD and major depression symptoms in military soldiers. Journal of Affective Disorders, 2015, 175:373-378.
[26] Gupta S, Goren A, Dong P, et al. Prevalence, awareness and burden of major depressive disorder in urban China. Value in Health, 2015, 18(3):A117.
[27] Evans S J, Choudary P V, Neal C R, et al. Dysregulation of the fibroblast growth factor system in major depression. Proceedings of the National Academy of Science of the United States of America, 2004, 101(43):15506-15511.
[28] Altmann C R, Brivanlou A H. Neural patterning in the vertebrate embryo. International Review of Cytology, 2001, 203(4):447-482.
[29] Briscoe J. Ericson J. Specification of neuronal fates in the ventral neural tube. Current Opinion in Neurobiology, 2001, 11(1):43-49.
[30] Liu A, Joyner A L. EN and GBX2 play essential roles downstream of FGF8 in patterning the mouse mid/hindbrain region. Development Cambridge, 2001, 128(1):181-191.
[31] Rhinn M, Brand M. The midbrain-hindbrain boundary organizer. Current Opinion in Neurobiology, 2001, 11(1):34-42.
[32] Simeone A. Towards the comprehension of genetic mechanisms controlling brain morphogenesis. Trends in Neurosciences, 2002, 25(3):119-121.
[33] Wilson S W, Brand M. Eisen J S. Patterning the zebrafish central nervous system. Results Probl Cell Differ. 2002, 40(40):181-215.
[34] Yaylaoglu M B, Titmus A, Visel A, et al. Comprehensive expression atlas of fibroblast growth factors and their receptors generated by a novel robotic in situ hybridization platform. Dev Dyn, 2005. 234(2):371-386.
[35] Sane J R, Yamagata M. Many paths to synaptic specificity. Annual Review of Cell and Developmental Biology, 2009, 25(25):161-195.
[36] Craig A M, Graf E R, Linhoff M W. How to build a central synapse:clues from cell culture. Trends in Neurosciences, 2006, 29(1):8-20.
[37] Fox M A, Umemori H. Seeking long-term relationship:axon and target communicate to organize synaptic differentiation. Journal of Neurochemistry, 2006, 97(5):1215-1231.
[38] Waites C L, Craig A M, Garner C C. Mechanisms of vertebrate synaptogenesis. Annual Review of Neuroscience, 2005, 28(1):251-274.
[39] Karami M, Bathaie S Z, Tiraihi T, et al. Crocin improved locomotor function and mechanical behavior in the rat model of contused spinal cord injury through decreasing calcitonin gene related peptide (CGRP). Phytomedicine, 2013, 21(1):62-67.
[40] Luís M C, José E P, Vítor M F, et al. Rolipram promotes functional recovery after contusive thoracic spinal cord injury in rats. Behavioural Brain Research, 2013, 243(3):66-67.
[41] Yang J R, Liao C H, Pang C Y, et al. Transplantation of porcine embryonic stem cells and their derived neuronal progenitors in a spinal cord injury rat model. Cytotherapy, 2013, 15(2):201-208.
[42] Marc Simard, Tsymbalyuk O, Keledjian K, et al. Comparative effects of glibenclamide and riluzole in a rat model of severe cervical spinal cord injury. Experimental Neurology, 2012, 233(1):566-574.
[43] Umemori H, Linhoff M W, Ornitz D M, et al. FGF22 and its close relatives are presynaptic organizing molecules in the mammalian brain. Cell, 2004, 118(2):257-270.
[44] Stevens H E, Smith K M, Maragnoli M E, et al. Fgfr2 is required for the development of the medial prefrontal cortex and its connections with limbic circuits. The Journal of Neuroscience, 2010, 30(16):5590-5602.
[45] Terauchi A, Johnson-Venkatesh E M, Toth A B, et al. Distinct FGFs promote differentiation of excitatory and inhibitory synapses. Nature, 2010, 465(7299):783-787.
[46] Jacobi A, Loy K, Schmalz A M, et al. FGF22 signaling regulates synapse formation during post-injury remodeling of the spinal cord. EMBO Journal, 2015, 34(9):1231-1243. |
