| [1] 齐金蕾, 王黎君, 周脉耕,等. 1990-2013年中国男性前列腺癌疾病负担分析. 中华流行病学杂志, 2016, 37(6):778-782. Qi J L, Wang L J, Zhou M G, et al. Disease burden of prostate cancer among men in China, from 1990 to 2013. Chinese Journal of Epidemiology, 2016,37(6):778-782.
[2] Wong M C, Goggins W B, Wang H H, et al. Global incidence and mortality for prostate cancer:analysis of temporal patterns and trends in 36 countries. Eur Urol, 2016, 70(5):862-874.
[3] Bergstrom S H, Rudolfsson S H, Bergh A. Rat prostate tumor cells progress in the bone microenvironment to a highly aggressive phenotype. Neoplasia, 2016, 18(3):152-161.
[4] Wellendorph P, Brauner-Osborne H. Molecular cloning, expression, and sequence analysis of GPRC6A, a novel family C G-protein-coupled receptor. Gene, 2004, 335(1):37-46.
[5] Clemmensen C, Smajilovic S, Wellendorph P, et al. The GPCR, class C, group 6, subtype A (GPRC6A) receptor:from cloning to physiological function. Br J Pharmacol, 2014, 171(5):1129-1141.
[6] Bolender D L, Markwald R R. Epithelial-mesenchymal transformation in chick atrioventricular cushion morphogenesis. Scan Electron Microsc, 1979, 3(3):313-321.
[7] Boyer B, Valles A M, Edme N. Induction and regulation of epithelial-mesenchymal transitions. Biochem Pharmacol, 2000, 60(8):1091-1099.
[8] Hanahan D, Weinberg R A. Hallmarks of cancer:the next generation. Cell, 2011, 144(5):646-674.
[9] Gonzalez R S, Huh W J, Cates J M, et al. Micropapillary colorectal carcinoma clinical, pathologic, and molecular properties, including evidence of epithelial-mesenchymal transition. Histopathology, 2016,70(2):223-231.
[10] Takata R, Akamatsu S, Kubo M, et al. Genome-wide association study identifies five new susceptibility loci for prostate cancer in the Japanese population. Nat Genet, 2010, 42(9):751-754.
[11] Lindström S, Schumacher F R, Campa D, et al. Replication of five prostate cancer loci identified in an Asian population——results from the NCI breast and prostate cancer cohort consortium (BPC3). Cancer Epidemiol Biomarkers Prev, 2012, 21(1):212-216.
[12] Wang M, Liu F, Hsing A W, et al. Replication and cumulative effects of GWAS-identified genetic variations for prostate cancer in Asians:a case-control study in the ChinaPCa consortium. Carcinogenesis, 2012, 33(2):356-360.
[13] Pi M, Quarles L D. GPRC6A regulates prostate cancer progression. Prostate, 2012, 72(4):399-409.
[14] 刘铭.中国北方人群前列腺癌风险位点识别和GPRC6A基因功能的研究.北京:北京协和医学院,2012. Liu M.Identification of prostate cancer risk SNPs in northern Chinese men and the functional study of GPRC6A gene. Beijing:Peking Union Medical College and Chinese Academy of Medical Sciences, 2012.
[15] Park J C, Eisenberger M A. Advances in the treatment of metastatic prostate cancer. Mayo Clin Proc, 2015, 90(12):1719-1733.
[16] Christiansen B, Hansen K B, Wellendorph P, et al. Pharmacological characterization of mouse GPRC6A, an L-alpha-amino-acid receptor modulated by divalent cations. Br J Pharmacol, 2007, 150(6):798-807.
[17] Pi M, Faber P, Ekema G, et al. Identification of a novel extracellular cation-sensing G-protein-coupled receptor. J Biol Chem, 2005, 280(48):40201-40209.
[18] Wang C, Liu Y, Cao J M. G protein-coupled receptors:extranuclear mediators for the non-genomic actions of steroids. Int J Mol Sci, 2014, 15(9):15412-15425.
[19] Gandalovicova A, Vomastek T, Rosel D, et al. Cell polarity signaling in the plasticity of cancer cell invasiveness. Oncotarget, 2016, 7(18):25022-25049.
[20] Lamouille S, Xu J, Derynck R. Molecular mechanisms of epithelial-mesenchymal transition. Nat Rev Mol Cell Biol, 2014, 15(3):178-196.
[21] Lee J Y, Kong G. Roles and epigenetic regulation of epithelial-mesenchymal transition and its transcription factors in cancer initiation and progression. Cell Mol Life Sci, 2016,73(24):4643-4660.
[22] Giannelli G, Koudelkova P, Dituri F, et al. Role of epithelial to mesenchymal transition in hepatocellular carcinoma. J Hepatol, 2016,65(4):798-808.
[23] Claperon A, Mergey M, Nguyen H T, et al. EGF/EGFR axis contributes to the progression of cholangiocarcinoma through the induction of an epithelial-mesenchymal transition. J Hepatol, 2014, 61(2):325-332.
[24] Peinado H, Olmeda D, Cano A. Snail, Zeb and bHLH factors in tumour progression:an alliance against the epithelial phenotype. Nat Rev Cancer, 2007, 7(6):415-428.
[25] Zhou B, Zhan H, Tin L, et al. TUFT1 regulates metastasis of pancreatic cancer through HIF1-Snail pathway induced epithelial-mesenchymal transition. Cancer Lett, 2016, 382(1):11-20. |
