[1] McNab F W, Rajsbaum R, Stoye J P, et al. Tripartite-motif proteins and innate immune regulation. Curr Opin Immunol, 2011, 23(1):46-56.
[2] Niida M, Tanaka M, Kamitani T. Downregulation of active IKK beta by Ro52-mediated autophagy. Mol Immunol, 2010, 47(14):2378-2387.
[3] Shi M, Deng W, Bi E, et al. TRIM30 alpha negatively regulates TLR-mediated NF-kappa B activation by targeting TAB2 and TAB3 for degradation. Nat Immunol, 2008, 9(4):369-377.
[4] 孙大康, 安新业, 郑静, 等. Trim5α对TAB2诱导的NFκB启动子荧光素酶报告基因活性的影响. 免疫学杂志, 2011, (07):580-584. Sun D K, An X Y, Zhen J, et al. Immunology, 2011, (07):580-584.
[5] Singh R, Gaiha G, Werner L, et al. Association of TRIM22 with the type 1 interferon response and viral control during primary HIV-1 infection. J Virol, 2011, 85(1):208-216.
[6] Ohmine S, Sakuma R, Sakuma T, et al. The antiviral spectra of TRIM5alpha orthologues and human TRIM family proteins against lentiviral production. PLoS One, 2011, 6(1):e16121.
[7] Tachado S D, Li X, Swan K, et al. Constitutive activation of phosphatidylinositol 3-kinase signaling pathway down-regulates TLR4-mediated tumor necrosis factor-alpha release in alveolar macrophages from asymptomatic HIV-positive persons in vitro. J Biol Chem, 2008, 283(48):33191-33198.
[8] Yarilina A, DiCarlo E, Ivashkiv L B. Suppression of the effector phase of inflammatory arthritis by double-stranded RNA is mediated by type I IFNs. J Immunol, 2007, 178(4):2204-2211.
[9] Mirandola S R, Hallal D E, Farias A S, et al. Interferon-beta modifies the peripheral blood cell cytokine secretion in patients with multiple sclerosis. Int Immunopharmacol, 2009, 9(7-8):824-830.
[10] Carthagena L, Bergamaschi A, Luna J M, et al. Human TRIM gene expression in response to interferons. PLoS One, 2009, 4(3):e4894.
[11] Rajsbaum R, Stoye J P, O'Garra A. Type I interferon-dependent and-independent expression of tripartite motif proteins in immune cells. Eur J Immunol, 2008, 38(3):619-630.
[12] Yoshimi R, Chang T H, Wang H, et al. Gene disruption study reveals a nonredundant role for TRIM21/Ro52 in NF-kappaB-dependent cytokine expression in fibroblasts. J Immunol, 2009, 182(12):7527-7538.
[13] Eldin P, Papon L, Oteiza A, et al. TRIM22 E3 ubiquitin ligase activity is required to mediate antiviral activity against encephalomyocarditis virus. J Gen Virol, 2009, 90(Pt 3):536-545.
[14] Gao B, Duan Z, Xu W, et al. Tripartite motif-containing 22 inhibits the activity of hepatitis B virus core promoter, which is dependent on nuclear-located RING domain. Hepatology, 2009, 50(2):424-433.
[15] Deng Y J, Huang Z X, Zhou C J, et al. Gene profiling involved in immature CD4+ T lymphocyte responsible for systemic lupus erythematosus. Mol Immunol, 2006, 43(9):1497-1507.
[16] Kim J Y, Ozato K. The sequestosome 1/p62 attenuates cytokine gene expression in activated macrophages by inhibiting IFN regulatory factor 8 and TNF receptor-associated factor 6/NF-kappaB activity. J Immunol, 2009, 182(4):2131-2140.
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