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[1] Grubman M J, Baxt B. Foot-and-mouth disease. Clin Microbiol Rev, 2004, 17(2): 465-493.
[2] Pelkmans L, Helenius A. Insider information: what viruses tell us about endocytosis. Curr Opin Cell Biol, 2003, 15(4): 414-422.
[3] Smith A E, Helenius A. How viruses enter animal cells. Science, 2004, 304(5668): 237-242.
[4] Barrow E, Nicola A V, Liu J. Multiscale perspectives of virus entry via endocytosis. Virol J, 2013, 10: 177.
[5] Hernaez B, Alonso C. Dynamin- and clathrin-dependent endocytosis in African swine fever virus entry. J Virol, 2010, 84(4): 2100-2109.
[6] Lee J J, Kim D G, Kim D H, et al. Interplay between clathrin and Rab5 controls the early phagocytic trafficking and intracellular survival of Brucella abortus within HeLa cells. J Biol Chem, 2013, 288(39): 28049-28057.
[7] O'Donnell V, Larocco M, Duque H, et al. Analysis of foot-and-mouth disease virus internalization events in cultured cells. J Virol, 2005, 79(13): 8506-8518.
[8] Berryman S, Clark S, Monaghan P, et al. Early events in integrin alpha v beta 6-mediated cell entry of foot-and-mouth disease virus. Journal of Virology, 2005, 79(13): 8519-8534.
[9] O'Donnell V, Pacheco J M, Larocco M, et al. Foot-and-mouth disease virus utilizes an autophagic pathway during viral replication. Virology, 2011, 410(1): 142-150.
[10] Du J, Chang H, Gao S, et al. Molecular characterization and expression analysis of porcine integrins alphavbeta3, alphavbeta6 and alphavbeta8 that are potentially involved in FMDV infection. Mol Cell Probes, 2010, 24(5): 256-265.
[11] Gullberg M, Muszynski B, Organtini L J, et al. Assembly and characterization of foot-and-mouth disease virus empty capsid particles expressed within mammalian cells. J Gen Virol, 2013, 94(Pt 8): 1769-1779.
[12] Ruiz-Saenz J, Goez Y, Tabares W, et al. Cellular receptors for foot and mouth disease virus. Intervirology, 2009, 52(4): 201-212.
[13] Bai X, Bao H, Li P, et al. Effects of two amino acid substitutions in the capsid proteins on the interaction of two cell-adapted PanAsia-1 strains of foot-and-mouth disease virus serotype O with heparan sulfate receptor. Virol J, 2014, 11: 132.
[14] Wang G, Wang Y, Shang Y, et al. How foot-and-mouth disease virus receptor mediates foot-and-mouth disease virus infection. Virol J, 2015, 12(1): 9.
[15] Burman A, Clark S, Abrescia N G A, et al. Specificity of the VP1 GH loo Pof foot-and-mouth disease virus for alpha v integrins. Journal of Virology, 2006, 80(19): 9798-9810.
[16] Berryman S, Clark S, Kakker N K, et al. Positively charged residues at the five-fold symmetry axis of cell culture-adapted foot-and-mouth disease virus permit novel receptor interactions. J Virol, 2013, 87(15): 8735-8744.
[17] Mohapatra J K, Pandey L K, Rai D K, et al. Cell culture adaptation mutations in foot-and-mouth disease virus serotype A capsid proteins: implications for receptor interactions. J Gen Virol, 2015, 96(Pt 3): 553-564.
[18] Mcmahon H T, Boucrot E. Molecular mechanism and physiological functions of clathrin-mediated endocytosis. Nat Rev Mol Cell Biol, 2011, 12(8): 517-533.
[19] Johns H L, Berryman S, Monaghan P, et al. A dominant-negative mutant of rab5 inhibits infection of cells by foot-and-mouth disease virus: implications for virus entry. J Virol,2009, 83(12): 6247-6256.
[20] Martin-Acebes M A, Gonzalez-Magaldi M, Sandvig K, et al. Productive entry of type C foot-and-mouth disease virus into susceptible cultured cells requires clathrin and is dependent on the presence of plasma membrane cholesterol. Virology, 2007, 369(1): 105-118.
[21] Butan C, Filman D J, Hogle J M. Cryo-electron microscopy reconstruction shows poliovirus 135S particles poised for membrane interaction and RNA release. J Virol, 2014, 88(3): 1758-1770.
[22] Organtini L J, Makhov A M, Conway J F, et al. Kinetic and structural analysis of coxsackievirus B3 receptor interactions and formation of the A-particle. J Virol, 2014, 88(10): 5755-5765.
[23] Tuthill T J, Harlos K, Walter T S, et al. Equine rhinitis A virus and its low pH empty particle: clues towards an aphthovirus entry mechanism? PLoS Pathog, 2009, 5(10): e1000620.
[24] O'Donnell V, Larocco M, Baxt B. Heparan sulfate-binding foot-and-mouth disease virus enters cells via caveola-mediated endocytosis. J Virol, 2008, 82(18): 9075-9085.
[25] Christianson H C, Belting M. Heparan sulfate proteoglycan as a cell-surface endocytosis receptor. Matrix Biol, 2014, 35: 51-55.
[26] Howes M T, Mayor S, Parton R G. Molecules, mechanisms, and cellular roles of clathrin-independent endocytosis. Curr Opin Cell Biol, 2010, 22(4): 519-527.
[27] Chaudhary N, Gomez G A, Howes M T, et al. Endocytic crosstalk: cavins, caveolins, and caveolae regulate clathrin-independent endocytosis. PLoS Biol, 2014, 12(4): e1001832.
[28] Bai X, Bao H, Li P, et al. Effects of two amino acid substitutions in the capsid proteins on the interaction of two cell-adapted PanAsia-1 strains of foot-and-mouth disease virus serotype O with heparan sulfate receptor. Virol J, 2014, 11: 132.
[29] Taylor M P, Kirkegaard K. Modification of cellular autophagy protein LC3 by poliovirus. J Virol, 2007, 81(22): 12543-12553.
[30] Lee Y R, Lei H Y, Liu M T, et al. Autophagic machinery activated by dengue virus enhances virus replication. Virology, 2008, 374(2): 240-248.
[31] Bird S W, Maynard N D, Covert M W, et al. Nonlytic viral spread enhanced by autophagy components. Proc Natl Acad Sci U S A, 2014, 111(36): 13081-13086.
[32] Berryman S, Brooks E, Burman A, et al. Foot-and-mouth disease virus induces autophagosomes during cell entry via a class III phosphatidylinositol 3-kinase-independent pathway. J Virol, 2012, 86(23): 12940-12953.
[33] Wong J, Zhang J, Si X, et al. Autophagosome supports coxsackievirus B3 replication in host cells. J Virol, 2008, 82(18): 9143-9153.
[34] Taylor M P, Kirkegaard K. Modification of cellular autophagy protein LC3 by poliovirus. J Virol, 2007, 81(22): 12543-12553.
[35] Lin L T, Dawson P W, Richardson C D. Viral interactions with macroautophagy: a double-edged sword. Virology, 2010, 402(1): 1-10.
[36] Xu Y, Eissa N T. Autophagy in innate and adaptive immunity. Proc Am Thorac Soc, 2010, 7(1): 22-28.
[37] Jackson W T, Giddings T J, Taylor M P, et al. Subversion of cellular autophagosomal machinery by RNA viruses. PLoS Biol, 2005, 3(5): e156.
[38] Steinberger J, Grishkovskaya I, Cencic R, et al. Foot-and-mouth disease virus leader proteinase: structural insights into the mechanism of intermolecular cleavage. Virology, 2014, 468-470: 397-408.
[39] Gladue D P, O'Donnell V, Baker-Branstetter R, et al. Foot-and-mouth disease virus nonstructural protein 2C interacts with Beclin1, modulating virus replication. J Virol, 2012, 86(22): 12080-12090.
[40] Wang J, Wang Y, Liu J, et al. A critical role of N-myc and STAT interactor (Nmi) in foot-and-mouth disease virus (FMDV) 2C-induced apoptosis. Virus Res, 2012, 170(1-2): 59-65.
[41] Knowles N J, Davies PR, Henry T, et al. Emergence in Asia of foot-and-mouth disease viruses with altered host range: characterization of alterations in the 3A protein. J Virol, 2001, 75(3): 1551-1556.
[42] Gladue D P, O'Donnell V, Baker-Bransetter R, et al. Interaction of foot-and-mouth disease virus nonstructural protein 3A with host protein DCTN3 is important for viral virulence in cattle. J Virol, 2014, 88(5): 2737-2747.
[43] Armer H, Moffat K, Wileman T, et al. Foot-and-mouth disease virus, but not bovine enterovirus, targets the host cell cytoskeleton via the nonstructural protein 3C(pro). Journal of Virology, 2008, 82(21): 10556-10566.
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